Items where Author is "Arch, Jonathan R.S."

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Number of items: 7.

Article

Wargent, Edward T. and Kępczyńska, Malgorzata A. and Zaibi, Mohamed S. and Hislop, David C. and Arch, Jonathan R.S. and Stocker, Claire J. (2020) High fat-fed GPR55 null mice display impaired glucose tolerance without concomitant changes in energy balance or insulin sensitivity but are less responsive to the effects of the cannabinoids rimonabant or Δ(9)-tetrahydrocannabivarin on weight gain. PeerJ. ISSN 2167-8359

Stocker, Claire J. and Wargent, Edward T. and Martin-Gronert, Malgorzata S and Cripps, Roselle L and Heisler, Lora K and Yeo, Giles S.H. and Ozanne, Susan E and Arch, Jonathan R.S. (2020) Developmental programming of appetite and growth in male rats increases hypothalamic serotonin (5-HT)5A receptor expression and sensitivity. International Journal of Obesity. ISSN 0307-0565

Trayhurn, Paul and Arch, Jonathan R.S. (2020) Is energy expenditure reduced in obese mice with mutations in the leptin/leptin receptor genes? Journal of Nutritional Science, 9 (e23). pp. 1-5.

Zaibi, Mohamed S. and Arch, Jonathan R.S. and Kępczyńska, Malgorzata A. and Wargent, Edward T. and Hislop, David C. and Eze, Dominic and Khedher, M.R.B. and Hammami, M. (2018) Preventive effects of Salvia officinalis leaf extract on insulin resistance and inflammation in a model of high fat diet-induced obesity in mice that responds to rosiglitazone. PeerJ. ISSN 2167-8359

Martin-Gronert, Malgorzata S and Stocker, Claire J. and Wargent, Edward T. and Cripps, Roselle L and Garfield, Alastair S and Jovanovic, Zorica and D'Agostino, Giuseppe and Yeo, Giles S.H. and Cawthorne, Michael A and Arch, Jonathan R.S. and Heisler, Lora K and Ozanne, Susan E (2016) 5-HT2A and 5-HT2C receptors as hypothalamic targets of developmental programming in male rats. Disease Models and Mechanisms. ISSN 1754-8411

Arch, Jonathan R.S. (2015) Horizons in the Pharmacotherapy of Obesity. Current Obesity Reports.

Arch, Jonathan R.S. and Trayhurn, Paul (2013) Detection of thermogenesis in rodents in response to anti-obesity drugs and genetic modification. Frontiers in Physiology, 4 (64). pp. 1-15.

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